Omega 3 & PUFA’s: What No One Wants To Talk About

The over-zealous, over-hyped allopathic, “one size fits all” approach to recommending Omega 3 oils is almost as obsessively rambunctious as the omega 6 corn and vegetable oil circus in the previous decades. Apparently the seed and fish oil industries have a vested interest in pumping out as many different kinds and brands of omega 3 oils as possible.

Omega 3 fatty acids are polyunsaturated oils. Polyunsaturated oils are referred to as PUFA’s (polyunsaturated fatty acids). PUFA’s are very unstable oils and as such not only have a high susceptibility to rancidity, but once consumed will result in heavy amounts of free radical activity in the human body as well as rapid and substantial loss of Vitamin E.

Back in the olden days, the “essential fatty acids” were considered arachadonic acid, linoleic acid and linoleic acids. Now that these omega 6 oils are known to have toxic effects in high amounts, the industry shifted its attention to the omega 3 variety of oils instead as being “essential”. Many serious researchers question whether or not there is such a thing as an “essential fatty acid”.

Omega 3 oils (Docosahexaenoic Acid, Eicosapentaenoic Acid, Alpha Linolenic Acid) are obtained exogenously from the diet. Their symptomatic benefit is due to their immunosuppressive inhibition of prostaglandin formation. Prostaglandins are fatty acid mediators which modulate various immune and inflammatory responses. Omega 3 fats block certain inflammatory prostaglandins. Their mechanism is very similar to how aspirin reduces pain symptoms.

It is important to point out that the short term anti-inflammatory, immunosuppressive effects that omega 3 fats have are due to the oxidized fats. While there are short term, anti-inflammatory benefits with omega 3, the oxidative damage that they induce can have long term consequences including lipofuscin (skin and organ age spots due to oxidative free radical activity), lipid peroxidative damage, Vitamin E loss and anti-mitochondrial effects.

Making Sense of Supplementing Omega 3

It makes sense to me that certain individuals could benefit from therapeutic, short term use of omega 3 fatty acids, while others can be harmed by their consumption. Anabolic hyperplasia is a condition which indicates an abnormal proliferation of cells. As the great Emanuel Revici, MD pointed out: “When there is only anabolism, there is only stasis. When there is only catabolism there is only chaos.” Famous cancer physicians such as Revici and Budwig used omega 3 fatty acids to treat cancer.

When anabolism predominates, cells are producing excessive amounts of lactic acid. Cancer begins as a result of anabolic hyperplasia. Omega 3 can be used in this regard to initiate catabolic disintegration of excessive anabolic activity.

However, someone with predominant catabolic conditions should minimize their intake of polyunsaturated oils. This is because people who are predominantly catabolic are already generating excess free radical activity.

In my opinion, omega 3 fatty acids should be recommended based upon individual metabolic necessity, something that mainstream allopathy does not address.